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§ɦṛɛɗɗịɛ ßịⱺ𝔩ⱺɠịᵴŧ

shreddy_scientist@lemmy.ml
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172 posts • 44 comments

“I would rather have questions that can’t be answered than answers that can’t be questioned.” - Rich Feynman

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uBlock doesn’t block cookies directly but it can be setup to block the domains associated with cookies. Adding the EasyList Cookie will accomplish this. It will prevent websites from setting cookies when you visit them. As far as essential cookies go, I’d assume the Easylist blocks cookies which won’t affect the websites function.

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What about the MullVad browser? I’m really liking it so far, been using it as my daily driver for a bit now, it’s rad!

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The fuck!? I’m already late for work, I don’t have time to Finish Him, or anyone! I got to get to the office!!

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It’s 100% a feature! Truly a horrendous approach to lock down a device someone purchased to the extent seen on Windows.

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Linux can’t prevent you from permanently removing files. While in Windows it’s a chore to remove a number of files/bloat, which are then most often back after a system update.

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Yep, @gravitas_deficiency@sh.itjust.works knows what’s up. PFAS are one group of forever chemicals. While 2,3,7,8-tetrachlorodibenzofuran (TCDF) shares many characteristics with “forever chemicals” like PFAS, including persistence, bioaccumulation, and toxicity, it belongs to a different chemical class (Persistent Organic Pollutants aka POPs). Nonetheless, its environmental and health impacts are significant, and efforts are needed to reduce its presence in the environment!

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This is another reason to favor Beyond over Impossible! Additionally, Beyond has always been gluten-free while Impossible made their recipe gluten-free years down the road.

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In part, according to one of the head researchers, “compared to prior studies that relied solely on epidemiological data, we integrated multiple layers of information, including epidemiological data, conventional metabolic biomarkers, and cutting-edge metabolomics”.

So in addition to finding the metadata, they also dug into the biomarkers present, which the molecular pathways are already known in detail. On top of this, metabolomics interpreted the chemical processes involving metabolites, the small molecule substrates, intermediates, and products of cell metabolism. Together, these aspects helped demonstrate the molecular consequences of ingesting animal products. The epidemiological layer assessed the incidence rate of developing T2D and then investigated how many involved the consumption of animal products for the individuals.

This being the case, it appears to be more than just correlation, as once the metadata was found, the team investigated the causation of the metadata’s findings.

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This is wild, TLR4 is associated with our immune system’s identification of LPS, which is found in the outer layer of gram-negative bacterial cells. So why it’s associated with the SARS-CoV-2 virus is mind-bending. For anyone interested in the TLR’s found in the immune system, here you go:

It’s well-known NF-kB is directly associated with NOD-Like-Receptors, which produces inflammation as an inflammasome. This is what allows immune cells to target the area in numbers. But the TLR4 involvement here is puzzling, would have 100% bet against its involvement in the immune response to SARS-CoV-2!

Edit: For clarity, S100 proteins are only found in vertebrates, and Heparan Sulfate is expressed by mammals on cell surfaces and in the surrounding extracellular matrix. HMGB1 works as a chromatin binding factor in our bodies. HSP60 + HSP70 (known as Heat Shock Proteins) are found in both prokaryotes and eukaryotes and are a response to stressful conditions. They’re basically chaperones and help control protein folding, transport, degradation, cell differentiation, and translocation. But viruses aren’t prokaryotes, so the HSPs they’re using during an infection are from the host. This helps show why I’m soo blown away with TLR4 being involved here!!

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I went with Petlibro for the feeder and fountain, they’ve worked well for me.

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